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dc.contributor.advisorTumor virusesen_US
dc.contributor.authorLudlow, Johnen_US
dc.contributor.authorSkuse, Garyen_US
dc.date.accessioned2006-08-29T13:59:42Zen_US
dc.date.available2006-08-29T13:59:42Zen_US
dc.date.issued1995-02en_US
dc.identifier.citationVirus Research 35N2 (1995) 113-121en_US
dc.identifier.issn0168-1702en_US
dc.identifier.urihttp://hdl.handle.net/1850/2549en_US
dc.description.abstractThe purpose of this review is to bring attention to some additional work in the tumor virus/tumor suppressor field which may have been overshadowed by reports describing adenovirus, SV40, and HPV oncoprotein binding to pRB and p53. The data reviewed herein provide further support for the model that a common mechanism by which DNA tumor viruses transform cells involves inactivation of cellular proteins which function as negative regulators of cell growth.en_US
dc.description.sponsorshipResearch in these laboratories is supported by NIH grants CA56940 (J.W.L.), CA55173 (G.R.S.), and Cancer Center Core Grant CA11198 awarded to the late Robert A. Cooper Jr.en_US
dc.format.extent37365 bytesen_US
dc.format.mimetypeapplication/pdfen_US
dc.language.isoen_USen_US
dc.publisherElsevier: Virus Researchen_US
dc.subjectOncoproteinsen_US
dc.subjectTumor suppressorsen_US
dc.titleViral oncoprotein binding to PRB, P107, P130, AND P300en_US
dc.typeArticleen_US
dc.identifier.urlhttp://dx.doi.org/10.1016/0168-1702(94)00094-S


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